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Bacon-Wrapped Scallops with Green Beans
Trapped Fat
Obesity pathogenesis as an intrinsic disorder in metabolic fuel partitioning
Front squat 3-3-3-3-3-3-3 reps
Tender scallops wrapped in smoky bacon, oven-roasted until crisp, and served alongside buttery garlic green beans.
Ingredients
For the Scallops:
12 large sea scallops (about 1 lb)
6 slices bacon, cut in half
2 Tbsp butter or tallow (melted, for brushing)
1 clove garlic, minced
½ tsp smoked paprika
Salt and black pepper, to taste
Toothpicks (for securing bacon)
For the Green Beans:
1 lb fresh green beans, trimmed
1 Tbsp butter or tallow
2 cloves garlic, minced
1 tsp lemon zest
Salt and black pepper, to taste
Optional Garnish:
Fresh parsley, chopped
Lemon wedges
Macronutrients
(per serving, makes 4)
Protein: 29g
Fat: 15g
Carbs: 12g
Preparation
Set the oven to 400°F and line a baking sheet with parchment paper.
Pat scallops dry and season lightly with salt and pepper. Wrap each scallop with half a slice of bacon, securing with a toothpick. Place on the baking sheet.
Mix melted butter with garlic and smoked paprika. Brush over scallops. Roast for 12–15 minutes, flipping halfway, until bacon is crisp and scallops are opaque (internal temp 125–130°F).
While scallops roast, heat butter in a skillet over medium. Add garlic, cook for 30 seconds, then toss in green beans. Sauté 4–5 minutes until tender-crisp. Season with lemon zest, salt, and pepper.
Arrange scallops on a platter with green beans. Garnish with parsley and lemon wedges.
Additionally, practice handstands for 20 minutes.
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In this 2024 review, the authors propose that obesity may be better understood not as a passive consequence of overeating, but as an intrinsic disorder of metabolic fuel partitioning in which fat becomes “trapped” in adipose tissue. Rather than excess calories simply spilling into fat stores, the paper argues that hormonal and cellular signals—particularly involving insulin and adipocyte biology—can impair fat mobilization, reducing the availability of circulating fuels and potentially driving hunger and energy conservation as compensatory responses. This model shifts the focus from willpower and caloric balance to the biological regulation of energy storage and release, suggesting that disrupted fuel trafficking may play a central role in obesity pathogenesis.
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