Join us for a Journal Club webinar with Bob Kaplan and Peter Shaw as they as they delve into the ROS Hypothesis of Obesity.

Rather than viewing all reactive oxygen species (ROS) as a negative responses, this article proposes that ROS aren’t just harmful byproducts of metabolism, but critical signals that regulate insulin sensitivity. We’ll explore how insulin actually relies on ROS to amplify glucose and fat uptake, how excess ROS feed back to limit further nutrient entry, and how different dietary fats shape this signaling.

The full article can be found here.

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